Why
short-term fasting
cures obesity helped by a low carb,
low fructose diet 8/9/16
Obesity is caused by a fat-
metabolism-appetite regulatory system in which insulin has the greatest influence;
thus to keep weight off fix the system. We
have 2 wrong messages 1) eat less fat thus more carbs and 2) eat less calories
and exercise more. The first causes obesity if carbs includes too much sugar,
and the second doesn’t work because if you are long-term overweight your
regulatory system has been set to its current weight, and that doesn’t change
with the calorie-restricted diet. Insulin
tells the cells to burn glucose and
thus store fat. The fix is to maintain low insulin thus fat burning which
starts when sleeping and continues with skipping
breakfast (I drink 16 ounce tea with tablespoon of lime juice). In the morning
I have plenty of energy, thus
feel good, am mentally sharp, in a good mood, and not very hungry. I do it every
morning now, because I like the way I feel.
The body stores very little glucose, thus while sleeping there is no
need for insulin to lower the blood glucose.
Without insulin, the cells switch to burning the stored fat. Staying
in fat burning longer allows the
liver to ship out some of the excess fat it has stored. The high sugar Western
diet has mess up the
liver to cause high insulin, also called insulin
resistance. Excess insulin causes excess fat storage in
the liver, muscles, fat, and pancreas cells.
Proteins, like glucose. causes insulin secretion; thus fat storage. Thus
a low carb diet needs the help of from short-term
fasting to fix the fatty liver.
Dr. Jason Fung in his clinic cures
his patients with fasting and a low carb diet.
As a nephrologist in the clinic he gets the worse patients, end stage
kidney failure from type-2 diabetes, on insulin injections, and thus they all
are quite obese. I have included in the
attachment a link to his lecture on YouTube and 2 blogs saved as word
documents. Because of 3 years of
full-time studies of diet related topics, I know that Dr. Fung is first among
scholars in the areas of obesity, diabetes, and the science relevant to their
causes and fixes (links at end of paper).
Hopefully, I have been sufficiently
convincing for you to open up the attachments and check out the evidence. One
paper is on what to eat and 5 diets
depending upon the situation, a long version with many additional topics, a
testimonial, and a Dr. Fung blog. Corporations
and the government that serves have been given us the wrong information on healthful
diet since 1977, low fats which are replaced with a high in sugar (thus
fructose) and other carbs. On
healthfully.org/rh are diet papers and much more. Feedback is always appreciated.
Extra part in attachment along with links:
Note: if you need a sweetener, buy
corn syrup (pure glucose). Several
grocery chains carry Karo syrup. Table sugar and corn syrup were the 2 main
sweeteners in the 1960s and before. The obesity
epidemic is caused by tripling the amount of fructose since the 1960s. Fructose
is found in fruits and is one-half of
the disaccharide sucrose (table
sugar), the other half is glucose.
Glucose can also form long types of chains (starches) that can be broken
down into single glucose units. These
long-chain easily digestible carbohydrates are found in plants, with the
highest concentration of them in grains, rice and potatoes. Fructose the other
half of sucrose is a net 15 times more reactive than glucose; it
attaches to proteins in a process called glycation
and this adversely affects
the functions of that protein. I have
included links to documentaries by the national broadcast corporations of
Canada and Australia on why too much sugar is poison—what U.S. corporate media
won’t play—it is the fructose half of sucrose.
If you dig deeper you will find out in a longer paper of mine (attached)
how through glycation on a high
sugar diet fructose is the principle cause for all of the conditions associated
with the western diet. I have at the
bottom of this paper links to my website pages and to a page with 130 links to
YouTube documentaries and lectures on an assortment of health issues, each with
a short description. Using Google Chrome,
I cast the YouTube to my television.
What has happened
to the
liver
The problem driving weight gain is
a fatty liver (see NAFLD in
Wikipedia, non-alcoholic fatty liver disease). Fatty
liver is a sign that reactive fructose has damaged proteins in the liver and
this resulted in raised insulin in the liver which caused excess fat storage in
the liver—this could have happened decades ago.
Note without excessive glycation by fructose, the liver doesn’t become
fatty. The traditional oriental diets are
very high in glucose (carbs); some are over 70%, yet their sugar which comes from
fruits and vegetables averages 15 grams daily.
Their liver is fine, thus obesity and type-2 diabetes are very
rare. Add Western levels of fructose to
their diet and they have the western health issues. Our Western diet in 2001
had 152 pounds of added sweeteners per person per year
according to the USDA (189 grams of added
sugars a day). Approximately half of
that is fructose. One sign of fatty
liver is visceral fat, and it doesn’t have to be very much. Some people
through vigilant weight control
put under 5 pounds around their middle (I am one of them). As described above
fructose goes to the
liver, the only place it is metabolized.
However, it is 15 times more reactive than glucose and attaches to
proteins in a process called “glycation”
(a bad thing). It is estimated that 1/3rd of
Europeans and Americans have NAFLD—I
think the percentage is at least twice, that the bar set for NAFLD is too high. NAFLD
messes up the complex functions of the liver including several that raise the
level of blood glucose, and thus cause the additional release of insulin from
the pancreas to lower the blood glucose. This is known as insulin
resistance. Prolonged high level of
insulin causes a gradual gain in weight.
And it gets worse, insulin also regulates leptin a hormone produced by
adipose (fat cells) which plays a key role in regulating fat storage. With
long-term weight gain this hormone adjusts to the larger fat cells, and it
operates to maintain that new level of fat.
Thus when that person attempts to lose weight on a low calorie diet, at
about 2 months leptin starts to lowers metabolism and increases hunger. This
effect of leptin will continue for years
after the weight is loss to restore the lost weight. With long-term weight,
this leptin system
resets to the new weight. Thus energy
restricted diets are doomed to fail (yo-yo diets) unless the liver is cleansed,
and this requires either bariatric surgery or fasting.
Profits before
people
Do
you wonder why this message about how fructose causes a fatty liver, the fasting
fix, that high insulin causes obesity, and
the role of fructose in the assortment of conditions including cardiovascular
disease, arthritis, type-2 diabetes, Alzheimer’s disease, macular degeneration,
isn’t on everyone’s mind, yet this evidence is pushed in peer review journal
articles? The answer is simple, the
media won’t offend its large advertisers, corporations to protect their market
influence the production of information (Dr. Fung’s article on Coke a Cola (see
attachment) and other companies fund
nutritional conferences--one of many way they influence information.
Drug companies do similar funding for physicians,
and our government is in bed with the corporate system that they once regulated
to protect the public. The evidence for
all my claims is supported by a large body of journal articles on bad pharma;
articles which meet the standards of peer review, yet the results are buried by
big-bucks corporate influence, their tobacco-science articles, and the use of
KOLs (key opinion leaders) for higher.
On my website I provide a few of the articles that support the claims I
have made on diet, my papers on diet have links to supporting journal articles,
and there is much more. I have put 12
years of university training and many hours into http://healthfully.org/rg/,
which has been up since 2004 and has over a thousand pages downloaded
daily.
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