Recommended Non-technical Dietary--JK

Home | Fat Metabolizing graph and explanation | Sugar white poison and the fix | Getting Started Fasting | Fasting is easy | Why Fasting, Low carbs Works | Why We Get Fat, and what to do about it | Why Some Don't Get Fat | Healing Obesity and Type-2 Diabetes: What To Do and Why | On Fructose causing Obesity and Diabetes and fix | Why We Get Fat, short + videos | Insulin resistance causes obesity & type 2 diabetes | Fung Long Distant Program info

Fat Metabolizing graph and explanation

Graph of fat-glucose metabolizing

Explanation  12/25/16 

The above graphs illustrate that there are two metabolic systems, the green burns fats; the orange burns glucose.  Insulin causes the body to burn glucose and thus to store fat.  The fix promoted by food manufacturers, pharma, corporate media, our government, and physicians is to “eat less and exercise more”.  This FAILS for the long-term overweight people because it doesn’t deal with the role of insulin and other hormones which regulate fat storage, and thus metabolism and appetite.  Only by extending the period in the green can the body repair the damage to this regulatory system.  This requires for the long-term overweight a low insulin diet with regular fasting. 


What went wrong, the fix: when to eat and what to eat

Like other mammals our weight, appetite, and metabolism is controlled by several dozen hormones.  The most important of these hormones are insulin and leptin.  The long-term obese and overweight have a damaged liver caused by too much of the sugar fructose.  Fructose is only metabolized in the liver. It is a net 15-times more reactive than glucose, where it damages the liver by a process called glycation, the random bonding to proteins.  This process is causal for 1) fatty liver, an excess 2-3 pounds of fat stored there; 2) insulin resistant, which entails that the level of insulin is higher than normal for the amount of blood glucose (sugar).  Since insulin causes the uptake from the blood of glucose for metabolism in cells, insulin also at the same time causes the cessation of fat metabolism and its storage as triglycerides.  Thus with higher than normal insulin there is a gradual increase in stored fat.  Insulin resistance and fatty liver causes the regulatory systems to make that person likely to have steady (yearly) weight gain.  If this excess fat accumulates for over a year or 2, the adipose (fat) tissue through its regulatory hormones will reset to its new level of fat.  Their energy restricted diet fails:  hormones slow metabolism and increase appetite to restore fat its prior amount.  See Dr. Fung’s The Complete Guide to Fasting, p. 101-6, on The Biggest Losers (TV show), who regained their weight.    

To succeed insulin resistance and fatty liver must be reverse, and this requires a very low insulin diet.  This diet metabolizes the excess fat without causing the regulatory hormones secreted by the fat tissue to hinder this process by reducing metabolism at least 25% (typically around 2-months on a low-calorie diet) and thereby creating a feeling that if that person ate more he would feel better and have more energy.  We also have inherited the mammalian system for having more energy to hunt and gather food when there is no food.  To utilize this system we merely have to go without eating.  Short term fasting, merely skipping breakfast and being on a low-carb, moderate-protein diet will succeed; If not than alternate day fasting works.  Fasting extends the period of fat burning (see graph) and eventually will cure insulin resistance and fatty liver disease (NAFLD).  Merely low carbs (ketogenic diet) often fails because incretin hormones in response to proteins causes insulin to be secreted.  Thus fasting is an essential part of the low-carb fix.   Once the goal is obtained most will only have to limit fructose[1] to under 20 grams a day, the lower the better. 

At are low-carb foods & low-insulin tables, dietary recommendations, and 5 diets according to goals; evidence for and advice on fasting at /rh/id13.html; fasting is easy at /rjk/id5.html; on fructose and glycation at /rjk/id1.html; which fats are good for you; and description of documentaries and lectures with link to YouTube at /rh/id7.html, which will confirm what I have just presented, and more.  

[1] The main sources of fructose are sucrose (table sugar) which is half fructose and half glucose, fruits, and high fructose corn syrup (HFCS), but not corn syrup and maltose which are pure glucose.  Fructose is a net 15 times more reactive than glucose, and it randomly attaches to proteins throughout the body to damage them and thus is the main cause for the age -related disease (RAGE) associated with this diet—including Alzheimer’s, heart disease, arthritis, etc. Starches found in rice, grains, and potatoes are glucose. The sugar galactose found in milk, but not cheeses, also should be limited since it is also very reactive and damages proteins. 

For the long-term overweight

Obesity is caused by a malfunctioning weight-appetite-metabolism regulatory system that causes the long-term increase in fat storage. As explained on the obverse side, the system malfunction when about 2 excess pounds of fat accumulates in the liver.  If the person has a long-term weight issue, their system has reset the normal level of fat to its higher level.  The fix is to metabolize more fat.  Fasting increase the amount of fat metabolized each day.  Their fix is to keep insulin low, since insulin causes cells to store fat.  This occurs through fasting and low carb diet.  Eventually their system will reset to the lower weight.  


The advice here is merely what JK would do in those situations. See your doctor for medical advice.

The only thing necessary for the triumph of evil is for good men to do nothing--Edmond Burke

Perverse systems grow by recruiting good men--jk

Everybody thinks they know what good is, but they are confused--Plato